A selected list of publications:
New tricks for an old slug: The critical role of postsynaptic mechanisms in learning and memory in Aplysia,
Prog. Brain Res,
Jami SA, Wright WG, Glanzman DL.
Differential classical conditioning of the gill-withdrawal reflex in Aplysia recruits both NMDA receptor-dependent enhancement and NMDA receptor-dependent depression of the reflex,
Glanzman, D. L
Simple minds: the neurobiology of invertebrate learning and memory,
Villareal G, Li Q, Cai D, Glanzman DL
The role of rapid, local, postsynaptic protein synthesis in learning-related synaptic facilitation in Aplysia,
The cellular mechanisms of learning in Aplysia: of blind men and elephants,
Li Q, Roberts AC, Glanzman DL.
Synaptic facilitation and behavioral dishabituation in Aplysia: dependence on release of Ca2+ from postsynaptic intracellular stores, postsynaptic exocytosis, and modulation of postsynaptic AMPA receptor efficacy,
Roberts AC, Glanzman DL.
Learning in Aplysia: looking at synaptic plasticity from both sides,
Ezzeddine Y, Glanzman DL.
Prolonged habituation of the gill-withdrawal reflex in Aplysia depends on protein synthesis, protein phosphatase activity, and postsynaptic glutamate receptors,
Chitwood RA, Li Q, Glanzman DL.
Serotonin facilitates AMPA-type reponses in isolated siphon motor neurons of Aplysia in culture,
2001; 534(Pt 2):
Murphy GG, Glanzman DL.
Cellular analog of differential classical conditioning in Aplysia: disruption by the NMDA receptor antagonist DL-2-amino-5-phosphonovalerate,