Jim Boulter, Ph.D.
Work Phone Number:
MRL Building, Room 2557
Los Angeles, CA 90095
MRL Building, Room 2774
Los Angeles, CA 90095
Psychiatry and Biobehavioral Sciences
Hatos Center for Neuropharmacology
Brain Research Institute
Dr. Jim Boulter is a Professor in the Department of Psychiatry and Biobehavioral Sciences. He received his doctorate degree in molecular biology from the University of California (Santa Barbara) and obtained postdoctoral training in molecular neuroscience at Dartmouth Medical School, the University of Texas (Houston), the University of California (San Diego), and the Salk Institute for Biological Studies. On the faculty of UCLA since 1996, Dr. Boulter's laboratory uses strains of genetically engineered mice to study the underlying cause of human inherited partial epilepsy and to characterize brain circuits involved in nicotine-induced dependence, tolerance, and withdrawal behaviors among habitual tobacco users. Dr. Boulter is a member of the UCLA Molecular Biology Institute, the Brain Research Institute, and the Hatos Research Center for Neuropharmacology.
Lioudyno, M. I. Verbitsky, M. Glowatzki, E. Holt, J. C. Boulter, J. Zadina, J. E. Elgoyhen, A. B. Guth, P. S.
The alpha9/alpha10-containing nicotinic ACh receptor is directly modulated by opioid peptides, endomorphin-1, and dynorphin B, proposed efferent cotransmitters in the inner ear
Mol Cell Neurosci,
Elgoyhen, A. B., Katz, E., Vetter, D. E., Rothlin, C. V., Heinemann, S., and Boulter, J
Alpha 10: A determinant of nicotinic cholinergic receptor function in mammalian vestibular and cochlear mechanosensory hair cells.,
Taranda Julian, Maison Stéphane F, Ballestero Jimena A, Katz Eleonora, Savino Jessica, Vetter Douglas E, Boulter Jim, Liberman M Charles, Fuchs Paul A, Elgoyhen A Belén
A point mutation in the hair cell nicotinic cholinergic receptor
prolongs cochlear inhibition and enhances noise protection.
Vetter, D. E. Katz, E. Maison, S. F. Taranda, J. Turcan, S. Ballestero, J. Liberman, M. C. Elgoyhen, A. B. Boulter, J.
The alpha10 nicotinic acetylcholine receptor subunit is required for normal synaptic function and integrity of the olivocochlear system
Proc Natl Acad Sci U S A,
Ladi, E. Nichols, J. T. Ge, W. Miyamoto, A. Yao, C. Yang, L. T. Boulter, J. Sun, Y. E. Kintner, C. Weinmaster, G.
The divergent DSL ligand Dll3 does not activate Notch signaling but cell autonomously attenuates signaling induced by other DSL ligands
J Cell Biol,
Ladi Ena, Nichols James T, Ge Weihong, Miyamoto Alison, Yao Christine, Yang Liang-Tung, Boulter Jim, Sun Yi E, Kintner Chris, Weinmaster Gerry
The divergent DSL ligand Dll3 does not activate Notch signaling but
cell autonomously attenuates signaling induced by other DSL ligands.
The Journal of cell biology,
Cui, C. Booker, T. K. Allen, R. S. Grady, S. R. Whiteaker, P. Marks, M. J. Salminen, O. Tritto, T. Butt, C. M. Allen, W. R. Stitzel, J. A. McIntosh, J. M. Boulter, J. Collins, A. C. Heinemann, S. F.
The beta3 nicotinic receptor subunit: a component of alpha-conotoxin MII-binding nicotinic acetylcholine receptors that modulate dopamine release and related behaviors
Cui Changhai, Booker T K, Allen Roberta S, Grady Sharon R, Whiteaker Paul, Marks Michael J, Salminen Outi, Tritto Theresa, Butt Christopher M, Allen W R, Stitzel Jerry A, McIntosh J Michael, Boulter Jim, Collins Allan C, Heinemann Stephen F
The beta3 nicotinic receptor subunit: a component of alpha-conotoxin
MII-binding nicotinic acetylcholine receptors that modulate dopamine
release and related behaviors.
The Journal of neuroscience : the official journal of the Society for Neuroscience,
Labarca, C Schwarz, J Deshpande, P Schwarz, S Nowak, MW Fonck, C Nashmi, R Kofuji, P Dang, H Shi, W Fidan, M Khakh, BS Chen, Z Bowers, BJ Boulter, J Wehner, JM Lester, HA
Point mutant mice with hypersensitive alpha 4 nicotinic receptors show dopaminergic deficits and increased anxiety.
Proceedings of the National Academy of Sciences of the United States of America. ,